Classification models of serial murderers have typically relied on
speculation, as the focus of serial murder, that the offender is driven to
murder due to his violent fantasies; suffers from some form of pathology; or
kills due to narcissistic drives. Another focus on serial
murderers that is propounded in the literature is that offenders kill to act
out sexual perversions. These types of crimes are defined as lust murders. There
are certainly sexual overtones to many serial murders, lust murderers may not
be representative of serial murderers at large. The
Facet Classification Model of serial murder does not carry direct implications
for the internal dynamics of serial murderers. The model proposes that more
objective accounts of what really happens in the course of a serial murder will
relate in part to the link between the offender and the victim. The objective
is important from a practical point of view, because it allows the study of the
material that is available in police records, which is within the realms of
what is referred to as Investigative Psychology. The basis
for developing a classification system of serial murder is the hypothesis that
variables derived from a crime scene will not have random interrelationships,
but rather will reveal a grouping of offenses that have consistently related
actions. There are a number of ways in which such association between the crime
scene behavior can be established, but whatever methods are used, they will be
more powerful in their application if they are part of a logical explanatory framework. For example, knowing which crime scene actions show
associations between offenses and offenders can be used to help classify behavior
into common themes. Implicit in the objective is the need to identify
behavioral traces at a crime scene which can be used as variables for this
research. Such traces of behavior may be seen as discrete acts, which
constitute one part, one element, or ingredient within a series of actions
which combine to form an underlying structure to a crime scene. that the analysis of the data will reveal the two
facets identified earlier, behavioral organization and attachment. The
behavioral facet will have two facet elements: affective and cognitive. As
previously mentioned, affective behavior is hypothesized to be manifested in
the form of erratic, emotional rage resulting in the offender’s crime scenes
being disorganized, while cognitive behavior is hypothesized to be manifested
in the form of organized, sadistic behavior. The second facet, attachment, also
has two elements: victim as vehicle and victim as object. Victim as vehicle is
most likely to be impersonal, suggesting that the offender’s interactions with
his victim is minimal, while victim as object is most likely to be personal,
suggesting that the offender imbues his victims with acts that require the
offender to spend long periods of time interacting with the victim’s body. The general
hypothesis consists of two groups:
1. there will be groups of serial murderers who will
consistently display signature behaviors that are more typical than any other
group, which will relate to background history, and;
2. that serial murderers will have certain crime scene
behaviors that they share in common.
Relating offender characteristics to offense is a
multivariate problem that has generally been ignored in the literature on
serial murder. It is appropriate to construe the relationships between offender
characteristics and offense as the canonical relationship between two matrixes:
the P matrix of offender characteristics and the Q matrix of offense behavior.
The P matrix is derived from the vectors generated by the attributes that
describe each offender (e.g., age and criminal history) and Q from the
attributes that describe each offense (e.g., type of weapon used and type of
sexual assault). In a situation in which an offender carries out one offense,
the P matrix is the same rank as the Q matrix and a direct mapping of one into
the other may be empirically feasible. However, in a situation in which an
offender carries out a series of murders, there is a one-to-many correspondence
between the vectors of P and Q that could lead to behavioral inconsistency
within an offense series and invalidate any extrapolation from offense to
offender. To solve this problem, the potential indeterminacy in resolving P>Q,
an iterative procedure, will be necessary whereby the empirical structure of
the P and Q matrixes are established independently. Dividing the overall data
sample into three subsamples allows for the exploration of consistency in
offenders’ crime scene behavior over time. The classification of crime scenes
by reference to their underlying themes implies that for each different theme
identified there will be a different type of offender, whose life experiences
will likely be different within various themes. Serial murderers found within
each theme will likely possess the same or parallel characteristics to those
offenders committing similar offenses. These data usually have no clear
existing structures and may be rather high in noise. The noise inherent in
real-world data stems from slight inconsistencies in the manner in which the
data were eventually collected.
Although most of the information collected by the
police is of a descriptive nature and not recorded in a quantifiable form, this
type of data still does have some advantages. First, since it is collected by
the police without any psychological hypotheses in mind, there could be less
bias in the data. Secondly, this type of data has ecological validity; it can
be seen in the context of the overall circumstances from where it was derived.
Acknowledgements:
The Police Department;
https://www.politie.nl/mijnbuurt/politiebureaus/05/burgwallen.html
and a Chief Inspector – Mr. Erik Akerboom
©
Bibliography:
1. Criminal
Investigations – Crime Scene Investigation.2000
2. Forensic
Science.2006
3. Techniques
of Crime Scene Investigation.2012
4. Forensics
Pathology.2001
5. Pathology.2005
6. Forensic
DNA Technology (Lewis Publishers,New York, 1991).
7. The
Examination and Typing of Bloodstains in the Crime Laboratory (U.S. Department
of Justice, Washington, D.C., 1971).
8. „A
Short History of the Polymerase Chain Reaction". PCR Protocols. Methods in
Molecular Biology.
9. Molecular
Cloning: A Laboratory Manual (3rd ed.). Cold Spring Harbor,N.Y.: Cold Spring
Harbor Laboratory Press.2001
10. "Antibodies as Thermolabile Switches: High Temperature
Triggering for the Polymerase Chain Reaction". Bio/Technology.1994
11. Forensic Science Handbook, vol. III (Regents/Prentice Hall,
Englewood Cliffs, NJ, 1993).
12. "Thermostable DNA Polymerases for a Wide Spectrum of
Applications: Comparison of a Robust Hybrid TopoTaq to other enzymes". In
Kieleczawa J. DNA Sequencing II: Optimizing Preparation and Cleanup. Jones and
Bartlett. 2006
13. Nielsen B, et al., Acute and adaptive responses in humans to
exercise in a warm, humid environment, Eur J Physiol 1997
14. Molnar GW, Survival of hypothermia by men immersed in the ocean.
JAMA 1946
15. Paton BC, Accidental hypothermia. Pharmacol Ther 1983
16. Simpson K, Exposure to cold-starvation and neglect, in Simpson K
(Ed): Modem Trends in Forensic Medicine. St Louis, MO, Mosby Co, 1953.
17. Fitzgerald FT, Hypoglycemia and accidental hypothermia in an
alcoholic population. West J Med 1980
18. Stoner HB et al., Metabolic aspects of hypothermia in the
elderly. Clin Sci 1980
19. MacGregor DC et al., The effects of ether, ethanol, propanol and
butanol on tolerance to deep hypothermia. Dis Chest 1966
20. Cooper KE, Hunter AR, and Keatinge WR, Accidental hypothermia.
Int Anesthesia Clin 1964
21. Keatinge WR. The effects of subcutaneous fat and of previous
exposure to cold on the body temperature, peripheral blood flow and metabolic
rate of men in cold water. J Physiol 1960
22. Sloan REG and Keatinge WR, Cooling rates of young people swimming
in cold water. J Appl Physiol 1973
23. Keatinge WR, Role of cold and immersion accidents. In Adam JM
(Ed) Hypothermia – Ashore and Afloat. 1981, Chapter 4, Aberdeen Univ. Press,
GB.
24. Keatinge WR and Evans M, The respiratory and cardiovascular
responses to immersion in cold and warm water. QJ Exp Physiol 1961
25. Keatinge WR and Nadel JA, Immediate respiratory response to
sudden cooling of the skin. J Appl Physiol 1965
26. Golden F. St C. and Hurvey GR, The “After Drop” and death after
rescue from immersion in cold water. In Adam JM (Ed). Hypothermia – Ashore and
Afloat, Chapter 5, Aberdeen Univ. Press, GB 1981.
27. Burton AC and Bazett HC, Study of average temperature of tissue,
of exchange of heat and vasomotor responses in man by means of bath
coloremeter. Am J Physiol 1936
28. Adam JM, Cold Weather: Its characteristics, dangers and
assessment, In Adam JM (Ed). Hypothermia – Ashore and Afloat, Aberdeen Univ.
Press, GB1981.
29. Modell JH and Davis JH, Electrolyte changes in human drowning
victims. Anesthesiology 1969
30. Bolte RG, et al., The use of extracorporeal rewarming in a child
submerged for 66 minutes. JAMA 1988
31. Ornato JP, The resuscitation of near-drowning victims. JAMA 1986
32. Conn AW and Barker CA: Fresh water drowning and near-drowning —
An update.1984;
33. Reh H, On the early postmortem course of “washerwoman’s skin at
the fingertips.” Z Rechtsmed 1984;
34. Gonzales TA, Vance M, Helpern M, Legal Medicine and Toxicology.
New York, Appleton-Century Co, 1937.
35. Peabody AJ, Diatoms and drowning – A review, Med Sci Law 1980
36. Foged N, Diatoms and drowning — Once more.Forens Sci Int 1983
37. "Microscale chaotic advection enables robust convective DNA
replication.". Analytical Chemistry. 2013
38. Sourcebook in Forensic Serology, Immunology, and Biochemistry
(U.S. Department of Justice, National Institute of Justice, Washington,
D.C.,1983).
39. C. A. Villee et al., Biology (Saunders College Publishing,
Philadelphia, 2nd ed.,1989).
40. Molecular Biology of the Gene (Benjamin/Cummings Publishing
Company, Menlo Park, CA, 4th ed., 1987).
41. Molecular Evolutionary Genetics (Plenum Press, New York,1985).
42. Human Physiology. An Integrate. 2016
43. Dumas JL and Walker N, Bilateral scapular fractures secondary to
electrical shock. Arch. Orthopaed & Trauma Surg, 1992; 111(5)
44. Stueland DT, et al., Bilateral humeral fractures from
electrically induced muscular spasm. J. of Emerg. Med. 1989
45. Shaheen MA and Sabet NA, Bilateral simultaneous fracture of the
femoral neck following electrical shock. Injury. 1984
46. Rajam KH, et al., Fracture of vertebral bodies caused by
accidental electric shock. J. Indian Med Assoc. 1976
47. Wright RK, Broisz HG, and Shuman M, The investigation of
electrical injuries and deaths. Presented at the meeting of the American
Academy of Forensic Science, Reno, NV, February 2000.
Komentarze
Prześlij komentarz