The FBI model
classifying offenders as disorganized, organized, or mixed was only the beginning of an effort to classify serial
killers. Part of the problem is that there is no precise definition of
insanity—doctors define insanity differently from courts of law. The
insanity plea has existed in English jurisprudence since the reign of Henry III
(1216–1272), when the king could commute a death sentence of an insane criminal
if it was demonstrated that irrational behaviour was not
unusual for the person in the past. In such cases, the prisoner would often end
up being confined in a monastery. In the next century the plea was moved
into the regular appeals process, no longer requiring the king’s
authority. In 1581 legal authorities were arguing what a test of insanity should consist of in
law, settling upon “knowledge of good and evil” as the test. In 1843,
English jurisprudence developed the concept of insanity as a defense against charges of murder. A mentally ill man
named Daniel M’Naghten came to believe that he was personally being
threatened by Prime Minister Sir Robert Peel and mistakenly shot and
killed Peel’s private secretary. He was acquitted on the grounds of insanity in
what is known to this day as the M’Naghten rule. And it says:
“It
must be clearly proved that, at the time of the committing of the act, the party
accused was laboring under such a defect of reason, from disease of the mind,
as not to know the nature and quality of the act he was doing; or, if he did
know it, that he did not know he was doing what was wrong”.
In other
words, to claim insanity, a defendant needs to prove that he could not distinguish between right and wrong, or that he
was not aware of what he was doing, because of mental illness. Trials where such a defense was
used required the conflicting testimony of psychiatrists who frequently
disagreed on an exact definition of “mental incapacity” and its effect on
an “irresistible impulse” of a defendant. Defense lawyers for serial
killers who were lucid, cleverly concealed their crimes, and functioned in their
daily lives argued that they were not guilty by reason of insanity—that they
could not resist the impulse to kill, even though they knew it was wrong,
because they were sick. The inability to resist an impulse to kill,
for whatever reason, is no longer a viable defense for a serial killer. Most serial
killers do not fit the legal definition of insanity, especially the organized ones.
They search for and stalk their victims, they arrive with weapons and
restraints prepared, they take their time killing, and they destroy the evidence
afterward, demonstrating a full knowledge of the consequences of their crime. They
elude detection and therefore are obviously aware of the wrongfulness of
their act. So what is wrong with these guys? Obviously something makes them
different from the rest of us. Describing them as merely evil seems
too simplistic and unscientific—after all, what is “evil” exactly?
Psychologists over the last decade have been desperately attempting to
evolve clinical definitions and explanations for the deeds and personalities of
serial killers. The two
concepts most often associated with serial killers by the public, press, law
enforcement, and academia are psychotic and psychopath. The two terms have very
different meanings. A psychotic is someone who has psychosis, a debilitating
organic mental illness, still not entirely understood, that can result in
delusions, hallucinations, and radical changes of behavior. Individuals with
this disorder are rarely violent, and very few serial killers are diagnosed as
psychotic. The psychotic’s incapacitating
disconnection with reality rarely makes him a good candidate for a long career
as a serial killer. Psychotics who display violence most often direct it at
themselves. On the other hand, serial killers
are most often diagnosed as psychopaths, or another closely related term,
sociopath. A psychopath is profoundly different from a psychotic. The
psychopathic state is not so much a mental illness as a behavioral or
personality disorder. One forensic psychiatrist described it as follows:
“A
morality that is not operating by any recognized or accepted moral code, but
operating entirely according to expediency to what one feels like doing at the
moment or that which will give the individual the most gratification or pleasure.
The term psychopathic state is the name we apply to those individuals who conform
to a certain intellectual standard, sometimes high, sometimes approaching the
realm of defect but yet not amounting to it, who throughout their lives, or
from a comparatively early age, have exhibited disorders of conduct of an
antisocial or asocial nature, usually of a recurrent or episodic type, where,
in many instances, have proved difficult to influence by methods of social,
penal, and medical care and treatment and for whom we have no adequate
provision of a preventive or curative nature. The inadequacy or deviation or
failure to adjust to ordinary social life is not a mere willfulness or badness
which can be threatened or thrashed out of the individual so involved, but
constitutes a true illness for which we have no specific explanation.”
Psychologists
theorize that psychopaths have a diminished capacity to experience fear and anxiety, which are the roots to the
normal development of a conscience. Psychopaths are often very charismatic
and very able at manipulating people. They are highly talented in
feigning emotions while inside feeling nothing. They have no remorse for
their victims and have highly developed psychological defense mechanisms
such as rationalization. (“She should have known better
than to hitchhike”), projection (“She was a heartless manipulating slut”), and
disassociation (“I don’t remember killing her”). They have a very weak
realization of self and compensate for that with grandiosity and an inflated
notion of entitlement— meaning that they feel that they are special and “entitled”
to act above the law or morality. Most notably, psychopaths lack any sense of
empathy with the feelings of others. A psychiatric lists some of the primary
characteristics of the psychopath, many if not all of which can be found in a
serial killer: glibness and superficial charm; grandiosity; continuous need for
stimulation; pathological lying; conning and manipulative behavior; lack of
remorse or guilt; shallow affect; callous lack of empathy; parasitic lifestyle;
poor behavioral controls; promiscuity; early behavior problems; lack of
realistic long-term goals; impulsivity; irresponsibility; failure to accept
responsibility for actions; many short-term relationships; juvenile
delinquency; revocation of conditional release; and criminal versatility. The
official psychiatric term for a psychopath is antisocial personality disorder. The
diagnostic criteria for this disorder, as described by the standard Diagnostic
and Statistical Manual of Mental Disorders, 4th edition (DSM-IV), are as
follows:
1. failure to conform to social norms with respect to
lawful behaviors as indicated by repeatedly performing acts that are grounds
for arrest;
2. deceitfulness, as indicated by repeated lying, use of
aliases, or conning others for personal profit or pleasure
3. impulsivity or failure to plan ahead
4. irritability and aggressiveness, as indicated by
repeated physical fights and assaults
5. reckless
disregard for the safety of self or others
6. consistent irresponsibility, as indicated by
repeated failure to sustain consistent work behaviour or honour financial obligations
7. lack of
remorse, as indicated by indifference to or rationalizing having hurt, mistreated, or stolen from another
Violent psychopaths emerge out of a combination of personal social conditions and biological and genetic
factors. Brain injuries can cause a psychopathic behavioural pattern; many serial
killers have a record of head injuries when they were children or
recent injuries prior to their beginning to kill. But this is not the
cause of their murderous behaviour—other behavioural problems are already present.
Healthy people who sustain head injuries do not become killers. Psychopaths also show abnormal balances of chemicals
currently linked to depression and compulsive behavior: monoamine oxidase (MAO)
and serotonin. They show cortical underarousal, high CSF free testosterone, and
EEG abnormalities. A high level of urine kryptopyrrole and an extra Y
chromosome have also been suspected as factors in the violent behavior of
psychopaths. There is evidence that some type
of congenital genetic abnormalities resulting in brain damage may be common to
many serial killers. Nineteenth century criminology tended to promote the idea
of a genetic criminal type. This idea has been swept away in the twentieth
century by environmental theories. Now criminology is beginning to steer a line
somewhere in between the two approaches. The prevailing theory is that there is a delicate
balance between a chaotic or abusive childhood and biochemical factors that can
trigger murderous psychopathic behavior. Healthy social factors can prevent a
biochemically unstable individual from committing criminal acts; healthy
biochemistry can protect a person with a turbulent childhood from growing up to
be a killer. Violent offenders emerge when both elements are out of balance.
This theory goes a long way to explain why some children with difficult
childhoods do not become serial killers and not everyone with a head injury behaves
criminally. The problem with psychopaths is
that the disorder is highly elusive— it is not a disease that can been directly
traced to a single chemical, viral, or organic agent; it is a mysterious
behavioral disorder whose history is buried deep in the offender’s psychology,
environment, and biochemistry. It is likely that a cure for cancer will be
found before psychopaths can be routinely treated and cured. Numerous serial
killers, after committing their first murders and being sent to psychiatric
facilities or receiving psychiatric treatment in prison, were deemed “cured”
and committed more escalated series of murders after their release. A psychiatric context is virtually impossible to definitively identify in cases of serial
murder. Serial killers seem to come close to a psychiatric definition of
psychopaths, but in the final analysis, they elude existing categories.
Acknowledgements:
The Police Department;
www.politie.nl and a Chief Inspector – Mr. Erik
Akerboom ©
Bibliography:
1. Criminal
Investigations – Crime Scene Investigation.2000
2. Forensic
Science.2006
3. Techniques
of Crime Scene Investigation.2012
4. Forensics
Pathology.2001
5. Pathology.2005
6. Forensic
DNA Technology (Lewis Publishers,New York, 1991).
7. The
Examination and Typing of Bloodstains in the Crime Laboratory (U.S. Department
of Justice, Washington, D.C., 1971).
8. „A Short
History of the Polymerase Chain Reaction". PCR Protocols. Methods in
Molecular Biology.
9. Molecular
Cloning: A Laboratory Manual (3rd ed.). Cold Spring Harbor,N.Y.: Cold Spring
Harbor Laboratory Press.2001
10. "Antibodies
as Thermolabile Switches: High Temperature Triggering for the Polymerase Chain
Reaction". Bio/Technology.1994
11. Forensic
Science Handbook, vol. III (Regents/Prentice Hall, Englewood Cliffs, NJ, 1993).
12. "Thermostable
DNA Polymerases for a Wide Spectrum of Applications: Comparison of a Robust
Hybrid TopoTaq to other enzymes". In Kieleczawa J. DNA Sequencing II:
Optimizing Preparation and Cleanup. Jones and Bartlett. 2006
13. Nielsen B,
et al., Acute and adaptive responses in humans to exercise in a warm, humid
environment, Eur J Physiol 1997
14. Molnar GW,
Survival of hypothermia by men immersed in the ocean. JAMA 1946
15. Paton BC,
Accidental hypothermia. Pharmacol Ther 1983
16. Simpson K,
Exposure to cold-starvation and neglect, in Simpson K (Ed): Modem Trends in
Forensic Medicine. St Louis, MO, Mosby Co, 1953.
17. Fitzgerald
FT, Hypoglycemia and accidental hypothermia in an alcoholic population. West J
Med 1980
18. Stoner HB
et al., Metabolic aspects of hypothermia in the elderly. Clin Sci 1980
19. MacGregor
DC et al., The effects of ether, ethanol, propanol and butanol on tolerance to
deep hypothermia. Dis Chest 1966
20. Cooper KE,
Hunter AR, and Keatinge WR, Accidental hypothermia. Int Anesthesia Clin 1964
21. Keatinge
WR. The effects of subcutaneous fat and of previous exposure to cold on the
body temperature, peripheral blood flow and metabolic rate of men in cold
water. J Physiol 1960
22. Sloan REG
and Keatinge WR, Cooling rates of young people swimming in cold water. J Appl
Physiol 1973
23. Keatinge
WR, Role of cold and immersion accidents. In Adam JM (Ed) Hypothermia – Ashore
and Afloat. 1981, Chapter 4, Aberdeen Univ. Press, GB.
24. Keatinge
WR and Evans M, The respiratory and cardiovascular responses to immersion in
cold and warm water. QJ Exp Physiol 1961
25. Keatinge
WR and Nadel JA, Immediate respiratory response to sudden cooling of the skin.
J Appl Physiol 1965
26. Golden F.
St C. and Hurvey GR, The “After Drop” and death after rescue from immersion in
cold water. In Adam JM (Ed). Hypothermia – Ashore and Afloat, Chapter 5,
Aberdeen Univ. Press, GB 1981.
27. Burton AC
and Bazett HC, Study of average temperature of tissue, of exchange of heat and
vasomotor responses in man by means of bath coloremeter. Am J Physiol 1936
28. Adam JM,
Cold Weather: Its characteristics, dangers and assessment, In Adam JM (Ed).
Hypothermia – Ashore and Afloat, Aberdeen Univ. Press, GB1981.
29. Modell JH
and Davis JH, Electrolyte changes in human drowning victims. Anesthesiology
1969
30. Bolte RG,
et al., The use of extracorporeal rewarming in a child submerged for 66
minutes. JAMA 1988
31. Ornato JP,
The resuscitation of near-drowning victims. JAMA 1986
32. Conn AW
and Barker CA: Fresh water drowning and near-drowning — An update.1984;
33. Reh H, On
the early postmortem course of “washerwoman’s skin at the fingertips.” Z
Rechtsmed 1984;
34. Gonzales
TA, Vance M, Helpern M, Legal Medicine and Toxicology. New York,
Appleton-Century Co, 1937.
35. Peabody
AJ, Diatoms and drowning – A review, Med Sci Law 1980
36. Foged N,
Diatoms and drowning — Once more.Forens Sci Int 1983
37. "Microscale
chaotic advection enables robust convective DNA replication.". Analytical
Chemistry. 2013
38. Sourcebook
in Forensic Serology, Immunology, and Biochemistry (U.S. Department of Justice,
National Institute of Justice, Washington, D.C.,1983).
39. C. A.
Villee et al., Biology (Saunders College Publishing, Philadelphia, 2nd
ed.,1989).
40. Molecular
Biology of the Gene (Benjamin/Cummings Publishing Company, Menlo Park, CA, 4th
ed., 1987).
41. Molecular
Evolutionary Genetics (Plenum Press, New York,1985).
42. Human Physiology.
An Integrate. 2016
43. Dumas JL
and Walker N, Bilateral scapular fractures secondary to electrical shock. Arch.
Orthopaed & Trauma Surg, 1992; 111(5)
44. Stueland
DT, et al., Bilateral humeral fractures from electrically induced muscular
spasm. J. of Emerg. Med. 1989
45. Shaheen
MA and Sabet NA, Bilateral simultaneous fracture of the femoral neck following
electrical shock. Injury. 1984
46. Rajam KH,
et al., Fracture of vertebral bodies caused by accidental electric shock. J.
Indian Med Assoc. 1976
47. Wright RK,
Broisz HG, and Shuman M, The investigation of electrical injuries and deaths.
Presented at the meeting of the American Academy of Forensic Science, Reno, NV,
February 2000.
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